They found that different spectral light stimulation, from infrared to white light, at one end of the spinal sensory or motor nerve roots resulted in a significant increase in the biophotonic activity at the other end, this is an evidence that biophotons can travel along neural fibers. Authors propose a protein–protein biophotonic interactions as mechanism for this conduction. In the experiments also tested the effects of procaine (an anesthetic) and of classic metabolic inhibitors, using both (anesthetic and metabolic inhibitor) cause quenched completely biophotonic activity, but using one solely don't suppress activity completely suggesting that the observed biophotonic signals after light stimulation consist of two components: action and background biophotons. It can be taken into account that procaine affects microtubule polymerization / depolymerization, and that microtubules are believed to act as optical fibers for biophoton propagation along axons. Anyways the authors propose a biophoton propagation through protein-protein interaction, where two different proteins may achieve biophotonic conduction if they form a biophotonic interaction couple, meaning that one protein absorbs a certain spectral biophoton (for example 630 nm) and emits another spectral biophoton (for example 670 nm). In contrast, the other protein of the couple absorbs 670 nm biophotons and emits 630 nm biophotons. In this way, 630 and 670 nm biophotons can conduct along a neural fiber if the protein couple is distributed and assembled in the neural fiber. On the other hand they remind us that there is experimental evidence for a correlation between biophotonic activity and the EEG activity. |
Last modified on 15-Mar-16 |